Cochlear mechanics · interactive model

Travelling Wave Lab

Basilar membrane motion, the outer hair cell amplifier and inner hair cell transduction for tones, clicks, tone bursts, two-tone pairs and speech, in normal and damaged cochleae. Time is slowed so you can follow each cycle.

Uncoiled cochlea: basilar membrane displacement

Base (stapes) on the left, apex (helicotrema) on the right. Human place–frequency map (Greenwood, 1990), 35 mm length. Dashed lines show the travelling-wave envelope.

BM displacement Envelope OHC health along the cochlea Probe place

Organ of Corti at the probe place

Radial cross-section. BM up (toward scala vestibuli) deflects stereocilia toward the tallest row and opens transduction channels.

Transduction at the probe place

Slowed time, most recent on the right.

Excitation pattern

Peak BM response (dB, model units) at every place. The dotted line is the level needed to drive the auditory nerve.

Input–output at probe CF

BM response to a tone at the probe's characteristic frequency.

What you are seeing

Clinical correlates

← ASLP Anatomy Studio · Pinna to Cortex (3D) · Concept and design: Hemaraja Nayaka S, Audiology and Speech-Language Pathology, Yenepoya Medical College Hospital, Mangaluru.

Model notes. This is a teaching model with realistic proportions, not a hydrodynamic simulation. Place map: Greenwood (1990), f = 165.4(100.06x − 0.88). OHC gain up to about 60 dB at mid–high CFs and about 35 dB near the apex, with about 0.2 dB/dB BM compression from 30 to 100 dB SPL (Ruggero et al., 1997). Loss of the active tip moves the passive peak about half an octave basally. IHC AC/DC ratio falls above about 1 kHz (Palmer & Russell, 1986). Phase locking weakens above about 2 kHz and is negligible by about 5 kHz. DPOAE settings are f2/f1 = 1.22 and L1 = L2 + 10 dB (Kummer et al., 1998). The dead-region concept and TEN(HL) test follow Moore (2004). Real OAE levels and travel times vary between individuals.